Oral

[3O10m-3] Alzheimer's Disease

Fri. Jul 31, 2020 11:10 AM - 12:10 PM Room 10

*Videos are available throughout the meeting period.

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[3O10m-3-01] YAP-dependent necrosis regulates early stages of Alzheimer’s disease pathology

*Hikari Tanaka¹, Hidenori Homma¹, Kyota Fujita¹, Kanoh Kondo¹, Xiaocen Jin¹, Kazuhiko Tagawa¹, Hitoshi Okazawa^1,2 (1. Dept. of Neuropathology, Medical Research Inst., Tokyo Medical and Dental Univ., 2. Center for Brain Integration Research, Tokyo Medical and Dental Univ.)

[3O10m-3-02] Diabetes mellitus exacerbates behavioral deficits and increases phosphorylation and aggregation of tau in Alzheimer's disease mouse model.

*Yuki Ito¹, Shuko Takeda¹, Tsuneo Nakajima², Akane Oyama², Hikari Takeshita², Yoichi Takami², Yasushi Takeya², Hiromi Rakugi², Ryuichi Morishita¹ (1. Dept Clinical Gene Therapy, Univ of Osaka, Osaka, Japan, 2. Dept Geriatric and General Medicine, Univ of Osaka, Osaka, Japan)

[3O10m-3-03] Fatty-acid-binding protein 3 is critical for α-Synuclein uptake and MPP⁺-induced mitochondrial dysfunction in dopaminergic neurons

*ICHIRO KAWAHATA¹, Luc Bousset², Ronald Melki², Kohji Fukunaga¹ (1. Dept. Pharm., Grad. Sch. Pharm. Sci., Tohoku Univ., 2. Lab. Neuro. Dis., CEA, MIRcen & CNRS)

[3O10m-3-04] A novel method for continuous collection of cerebrospinal fluid in the free-behaving mice for a preclinical biomarker research.

*Tsuneo Nakajima¹, Shuko Takeda², Akane Oyama¹, Yuki Ito², Youichi Takami¹, Yasushi Takeya¹, Ken Sugimoto¹, Koichi Yamamoto¹, Hiromi Rakugi¹, Ryuichi Morishita² (1. Geriatric and General Medicine, Osaka University Graduate School of Medicine, 2. Department of Clinical Gene Therapy, Graduate School of Medicine, Osaka University)

Session information

[3O10m-3] Alzheimer's Disease

[3O10m-3-01] YAP-dependent necrosis regulates early stages of Alzheimer’s disease pathology

[3O10m-3-02] Diabetes mellitus exacerbates behavioral deficits and increases phosphorylation and aggregation of tau in Alzheimer's disease mouse model.

[3O10m-3-03] Fatty-acid-binding protein 3 is critical for α-Synuclein uptake and MPP⁺-induced mitochondrial dysfunction in dopaminergic neurons

[3O10m-3-04] A novel method for continuous collection of cerebrospinal fluid in the free-behaving mice for a preclinical biomarker research.

Authentication

Session information

[3O10m-3] Alzheimer's Disease

[3O10m-3-01] YAP-dependent necrosis regulates early stages of Alzheimer’s disease pathology

[3O10m-3-02] Diabetes mellitus exacerbates behavioral deficits and increases phosphorylation and aggregation of tau in Alzheimer's disease mouse model.

[3O10m-3-03] Fatty-acid-binding protein 3 is critical for α-Synuclein uptake and MPP+-induced mitochondrial dysfunction in dopaminergic neurons

[3O10m-3-04] A novel method for continuous collection of cerebrospinal fluid in the free-behaving mice for a preclinical biomarker research.

Authentication

[3O10m-3-03] Fatty-acid-binding protein 3 is critical for α-Synuclein uptake and MPP⁺-induced mitochondrial dysfunction in dopaminergic neurons