*Gina Griffioen-Järfalk1,2, Martin Bolin1, Vladimir Stephanov1, Simon Cervenka1,6, Miguel Castelo-Branco7, Ricardo Martins7, Johan Lundberg1, Chirag Mehra8, Oliver Howes8, Declan Murphy8, Sven Bölte1,3,4,5, Lars Farde1, Jacqueline Borg1,2
(1. 1.Department of Clinical Neuroscience, Center for Psychiatry Research, Karolinska Institutet and Region Stockholm, Stockholm, Sweden, 2. 2.Center for Cognitive and Computational Neuropsychiatry (CCNP), Karolinska Institutet, Stockholm, Sweden, 3. 3.Department of Women’s and Children’s Health, Karolinska Institutet, Center of Neurodevelopmental Disorders (KIND), Stockholm, Sweden, 4. 4.Child and Adolescent Psychiatry, Stockholm Health Care Services, Region Stockholm, Stockholm, Sweden, 5. 5.Curtin Autism Research Group, School of Occupational Therapy, Social Work and Speech Pathology, Curtin University, Perth, WA, Australia, 6. 6. Department of Medical Sciences, Psychiatry, Uppsala University, Uppsala, Sweden, 7. Coimbra Institute for Biomedical Imaging and Translational Research, University of Coimbra, Portugal, 8. Institute of Psychiatry, Psychology and Neuroscience, King's College London, UK)
Keywords:Autism, synaptopathology, Positron Emission Tomography, connectivity, twin study
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