IP3R2, a contact site component on MAMs, alters proximal tubular cell arrest and survival caused by the anoxia-reperfusion injury

Qi Li

Presentation information

一般演題口演

一般演題口演 AKI（基礎）2

Sat. Jun 29, 2024 2:00 PM - 3:00 PM 第10会場 (ノース 4階 G412+G413)

司会：三村維真理(東京大学医学部附属病院腎臓・内分泌内科)

[O-210] IP3R2, a contact site component on MAMs, alters proximal tubular cell arrest and survival caused by the anoxia-reperfusion injury

^○Qi Li¹, Sho Hasegawa¹, Yuto Takenaka¹, Chigusa Kitayama¹, Kenzo Hirose², Shigeyuki Namiki², Masaomi Nangaku¹, Reiko Inagi¹ (1.Division of Nephrology and Endocrinology, The University of Tokyo, 2.Department of Pharmacology, The University of Tokyo)

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