一般演題口演 AKI（基礎）2

[O-210] IP3R2, a contact site component on MAMs, alters proximal tubular cell arrest and survival caused by the anoxia-reperfusion injury

^○Qi Li¹, Sho Hasegawa¹, Yuto Takenaka¹, Chigusa Kitayama¹, Kenzo Hirose², Shigeyuki Namiki², Masaomi Nangaku¹, Reiko Inagi¹ (1.Division of Nephrology and Endocrinology, The University of Tokyo, 2.Department of Pharmacology, The University of Tokyo)