The novel mice model to investigate the mechanisms of Glial cytoplasmic inclusions (GCIs) formation in multiple system atrophy (MSA)

Tomoyuki Ishimoto

[1P-212] The novel mice model to investigate the mechanisms of Glial cytoplasmic inclusions (GCIs) formation in multiple system atrophy (MSA)

*Tomoyuki Ishimoto¹, Hodaka Yamakado¹, Miki Oono¹, Seiji Kaji¹, Takashi Ayaki¹, Takakuni Maki¹, Shu-ichi Matsuzawa¹, Ryosuke Takahashi¹ (1. Department of Neurology Kyoto University Graduate School of Medicine)

Keywords:Multiple system atrophy, alpha synuclein, oligodendrocyte, Glial cytoplasmic inclusions

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PW: Neuro2022

Presentation information

[1P] F. Disorders of the Nervous System 2

[1P-212] The novel mice model to investigate the mechanisms of Glial cytoplasmic inclusions (GCIs) formation in multiple system atrophy (MSA)

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