[2Pa] Neurodegenerative Disorders

Wed. Aug 2, 2023 4:20 PM - 5:20 PM Poster Room (Exhibition Building)

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[2Pa-104] Mitochondrial dysfunction in a mouse model of
prodromal Parkinson’s disease: metabolomic
analysis

○Masashi Ikuno Ikuno¹, Hodaka Yamakado¹, Ryosuke Takahashi¹ (1. Kyoto University)

[2Pa-105] Specific administration of a novel anti-IL-18 antibody during effector phase improves EAE

○Yuki Nakashima¹, Hisao Miyajima¹, Shota Sasaki¹, Huy Xuan Ngo², Yoshinori Otani¹, Masashi Fujitani¹ (1. Department of Anatomy and Neuroscience, Faculty of Medicine, Shimane University, 2. Department of Oral and Maxillofacial Surgery, Faculty of Medicine, Shimane University)

[2Pa-106] Pathophysiological exploration of motor neuron vulnerability in ALS with focus on axons using iPS cells with TARDBP mutations.

○Shio Mitsuzawa^1,2, Naoki Suzuki¹, Hitoshi Warita¹, Hideyuki Okano³, Masashi Aoki¹ (1. Tohoku University Graduate School of Medicine, 2. Department of Neurology, Shodo-kai Southern TOHOKU General Hospital, 3. Department of Physiology, School of Medicine, Keio University)

[2Pa-107] TDP-43 differentially induces antero- and retrograde degeneration in the corticospinal circuits in mouse ALS models

○Shintaro Tsuboguchi¹, Yuka Nakamura¹, Tomohiko Ishihara¹, Taisuke Kato¹, Tokiharu Sato¹, Akihide Koyama², Hideki Mori¹, Yuka Koike¹, Osamu Onodera¹, Masaki Ueno¹ (1. Brain Research Institute, Niigata University, 2. Graduate School of Medicine and Dental Sciences, Niigata University )

[2Pa-108] Sigma-1 receptor maintains ATAD3A as a monomer to prevent mitochondrial fragmentation in amyotrophic lateral sclerosis

○Seiji Watanabe¹, Mai Horiuchi¹, Koji Yamanaka¹ (1. RIEM, Nagoya Univ, Nagoya, Japan)

Session information

[2Pa] Neurodegenerative Disorders

[2Pa-104] Mitochondrial dysfunction in a mouse model of
prodromal Parkinson’s disease: metabolomic
analysis

[2Pa-105] Specific administration of a novel anti-IL-18 antibody during effector phase improves EAE

[2Pa-106] Pathophysiological exploration of motor neuron vulnerability in ALS with focus on axons using iPS cells with TARDBP mutations.

[2Pa-107] TDP-43 differentially induces antero- and retrograde degeneration in the corticospinal circuits in mouse ALS models

[2Pa-108] Sigma-1 receptor maintains ATAD3A as a monomer to prevent mitochondrial fragmentation in amyotrophic lateral sclerosis

Authentication

Session information

[2Pa] Neurodegenerative Disorders

[2Pa-104] Mitochondrial dysfunction in a mouse model ofprodromal Parkinson’s disease: metabolomicanalysis

[2Pa-105] Specific administration of a novel anti-IL-18 antibody during effector phase improves EAE

[2Pa-106] Pathophysiological exploration of motor neuron vulnerability in ALS with focus on axons using iPS cells with TARDBP mutations.

[2Pa-107] TDP-43 differentially induces antero- and retrograde degeneration in the corticospinal circuits in mouse ALS models

[2Pa-108] Sigma-1 receptor maintains ATAD3A as a monomer to prevent mitochondrial fragmentation in amyotrophic lateral sclerosis

Authentication

[2Pa-104] Mitochondrial dysfunction in a mouse model of
prodromal Parkinson’s disease: metabolomic
analysis