[1P-091（2T07a-08）] Role of transmembrane protein 168 mutations in Brugada syndrome by alteration in subcellular localization of αB-crystallin to modulate sodium channel Na _v1.5 surface expression in cardiomyocytes

^〇Nguyen Le Kim Chi¹, Shimizu Akio ¹, Sato Akira ¹, Ogita Hisakazu¹ (Division of Molecular Medical Biochemistry, Department of Biochemistry and Molecular Biology, Shiga University of Medical Science)

キーテクノロジー：immunostainning,western blot

Transmembrane protein 168、Brugada syndrome、α B-crystallin、cardiac sodium channel