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[2T07a-08(1P-091)] Role of transmembrane protein 168 mutations in Brugada syndrome by alteration in subcellular localization of αB-crystallin to modulate sodium channel Na v1.5 surface expression in cardiomyocytes

Nguyen Le Kim Chi1, Akio Shimizu1, Akira Sato1, Hisakazu Ogita1 (Division of Molecular Medical Biochemistry, Department of Biochemistry and Molecular Biology, Shiga University of Medical Science)

キーテクノロジー:immunostainning,western blot

Transmembrane protein 168、Brugada syndrome、α B-crystallin、cardiac sodium channel

bschikim@gmail.com

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