The Japanese Biochemical Society

[3P-161] Rab32 and Rab38 facilitate osteoclast function by regulating the intracellular traffic of acid hydrolases and V-ATPase

Kanako Tokuda1, Shiou-Ling Lu2, Siyu Chen2, Kazuya Noda2,3, Katsutoshi Hirose4, Yu Usami4, Shinya Murakami3, Satoru Toyosawa4, Mitsunori Fukuda5, Ge-Hong Sun-Wada6, Yoh Wada7, Takeshi Noda1,2 (1.Graduate School of Frontier Biosciences, Osaka University, 2.Center for Frontier Oral Science, Graduate School of Dentistry, Osaka University, 3.Department of Periodontology, Graduate School of Dentistry, Osaka University, 4.Department of Oral Pathology, Osaka University Graduate School of Dentistry , 5.Graduate School of Life Sciences, Tohoku University, 6.Faculty of Pharmaceutical Sciences, Doshisha Women's College, 7.Institute of Scientific and Industrial Research, Osaka University,)

キーテクノロジー:Crispr-Cas9 Double KO mouse

Rab32/Rab38、破骨細胞、骨吸収

lu.shiouling.dent@osaka-u.ac.jp

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