α2-antiplasmin is a key regulator of macrophage activation syndrome progression through modulation of IFN-γ-induced responses and fibrin deposition.

Yosuke Kanno

[1P-539] α2-antiplasmin is a key regulator of macrophage activation syndrome progression through modulation of IFN-γ-induced responses and fibrin deposition.

^〇Yosuke Kanno¹, Kinomi Toyama¹, Haruna Shibata¹, Osamu Matsuo², Kei-ichi Ozaki¹ (1.Department of Molecular Pathology, Faculty of Pharmaceutical Science, Doshisha Womens College of Liberal Arts, 2.Faculty of Medicine, Kindai University)

キーテクノロジー：病態解析

線維化、マクロファージ活性化症候群、α2-antiplasmin、fibrin

Contents of this site has moved to the member page of The Japanese Biochemical Society: https://member.jbsoc.or.jp/mypage/
You can access the abstracts without credentials by accessing them via the member page.

You need the membership number and password to access the member page. Please contact the journal office for any inquiries.

Journal Office Contact Information:
E-mail:jbs-ho@jbsoc.or.jp
Tel:03(3815)1913 Fax:03(3815)1934

The 96th Annual Meeting of the Japanese Biochemical Society

[1P-539] α2-antiplasmin is a key regulator of macrophage activation syndrome progression through modulation of IFN-γ-induced responses and fibrin deposition.

Password