Rab32 and Rab38 facilitate osteoclast function by regulating the intracellular traffic of acid hydrolases and V-ATPase.

Kanako Tokuda

[2P-287] Rab32 and Rab38 facilitate osteoclast function by regulating the intracellular traffic of acid hydrolases and V-ATPase.

Kanako Tokuda¹, ^〇Shiou-Ling Lu², Siyu Chen², Katuya Noda^2,3, Katsutoshi Hirose⁴, Yu Usami⁴, Shinya Murakami³, Satoru Toyosawa⁴, Mitsunori Fukuda⁵, Ge-Hong Sun-Wada⁶, Yoh Wada⁷, Takeshi Noda^1,2 (1.Graduate School of Frontier Biosciences, Osaka University, 2.Center for Frontier Oral Science, Graduate School of Dentistry, Osaka University, 3.Department of Periodontology, Graduate School of Dentistry, Osaka University, 4.Department of Oral Pathology, Graduate School of Dentistry, Osaka University , 5.Graduate School of Life Sciences, Tohoku University, 6.Faculty of Pharmaceutical Sciences, Doshisha Women's College, 7.Institute of Scientific and Industrial Research, Osaka University)

キーテクノロジー：Crispr-Cas9 Double KO mouse

破骨細胞、Rab32/Rab38、骨吸収

lu.shiouling.dent@osaka-u.ac.jp

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The 96th Annual Meeting of the Japanese Biochemical Society

[2P-287] Rab32 and Rab38 facilitate osteoclast function by regulating the intracellular traffic of acid hydrolases and V-ATPase.

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