[2P-300] Phosphorylation of phase-separated p62 bodies by ULK1 activates a redox-independent stress response

^〇Yoshinobu Ichimura¹, Ryo Ikeda^1,2, Daisuke Noshiro³, Hideaki Morishita^1,8, Shuhei Takada¹, Shun Kageyama¹, Yuko Fujioka³, Tomoko Funakoshi¹, Satoko Komatsu-Hirota¹, Ritsuko Arai⁴, Elena Ryzhii⁴, Manabu Abe⁵, Tomoaki Koga⁶, Hozumi Motohashi⁷, Mitsuyoshi Nakao⁶, Kenji Sakimura⁵, Arata Horii², Satoshi Waguri⁴, Nobuo N. Noda³, Masaaki Komatsu¹ (1.Department of Physiology, Juntendo University Graduate School of Medicine, 2.Department of Otolaryngology Head and Neck Surgery, Niigata University Graduate School of Medical and Dental Sciences, 3.Institute for Genetic Medicine, Hokkaido University, 4.Department of Anatomy and Histology, Fukushima Medical University School of Medicine, 5.Department of Animal Model Development, Brain Research Institute, Niigata University, 6.Department of Medical Cell Biology, Institute of Molecular Embryology and Genetics, Kumamoto University, 7.Department of Gene Expression Regulation, Institute of Development, Aging and Cancer, Tohoku University, 8.Graduate School of Medical Sciences, Kyushu University)

キーテクノロジー：高速原子間力顕微鏡

p62、ULK1、リン酸化、液―液相分離、KEAP1-NRF2経路