Palmitic acid induced insulin resistance via disruption of the contact site between the ER and mitochondria, without involvement of ER stress response and ROS.

Satoko Shinjo; Nobuhito Goda

[1P-0287] Palmitic acid induced insulin resistance via disruption of the contact site between the ER and mitochondria, without involvement of ER stress response and ROS.

〇Satoko Shinjo¹, Nobuhito Goda¹ (1.Dept. of Life Sci. and Med. Biosci., Sch. of Adv. Sci. and Eng., Waseda Univ.)

mitochondria associated membrane, insulin resistance, type 2 diabetes

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The 39th Annual Meeting of the Molecular Biology Society of Japan

[1P-0287] Palmitic acid induced insulin resistance via disruption of the contact site between the ER and mitochondria, without involvement of ER stress response and ROS.

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