Transmembrane protein 240 (TMEM240), a causative protein of spinocerebellar ataxia type 21, triggers vesicular formation and impairment of autophagic-lysosomal protein degradation

Takahiro Seki; Masahiro Sato; Mutsumi Oshima; Yuki Kibe; Ayumu Konno; Hirokazu Hirai; Yuki Kurauchi; Akinori Hisatsune; Hiroshi Katsuki

[1P-0551] Transmembrane protein 240 (TMEM240), a causative protein of spinocerebellar ataxia type 21, triggers vesicular formation and impairment of autophagic-lysosomal protein degradation

〇Takahiro Seki¹, Masahiro Sato¹, Mutsumi Oshima¹, Yuki Kibe¹, Ayumu Konno², Hirokazu Hirai², Yuki Kurauchi¹, Akinori Hisatsune^3,4, Hiroshi Katsuki¹ (1.Dept. Chemico-Pharmacol. Sci., Grad. Sch. Pharm. Sci., Kumamoto Univ., 2.Dept. Neurophysiol. Neural Repear, Gunma Univ. Grad. Scho. Med., 3.Priority Organization for Innovation and Excellence, Kumamoto Univ., 4.Program for Leading Grad. Sch. "HIGO Program", Kumamoto Univ.)

spinocerebellar ataxia type 21, TMEM240, macroautophagy, microautophagy, chaperone-mediated autophagy

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The 39th Annual Meeting of the Molecular Biology Society of Japan

[1P-0551] Transmembrane protein 240 (TMEM240), a causative protein of spinocerebellar ataxia type 21, triggers vesicular formation and impairment of autophagic-lysosomal protein degradation

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