Gain-of-function USP8 mutations in Cushing's disease cause the stabilization of vasopressin receptor V1b that promotes ACTH secretion

Toshiaki Fukushima; Kohei Kawaguchi; Akinori Endo; Masayuki Komada

[2P-0789] Gain-of-function USP8 mutations in Cushing's disease cause the stabilization of vasopressin receptor V1b that promotes ACTH secretion

〇Toshiaki Fukushima¹, Kohei Kawaguchi¹, Akinori Endo¹, Masayuki Komada¹ (1.Cell Biol. Unit, Inst. of Innov. Res., Tokyo Tech)

Cushing's disease, USP8, vasopressin

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The 39th Annual Meeting of the Molecular Biology Society of Japan

[2P-0789] Gain-of-function USP8 mutations in Cushing's disease cause the stabilization of vasopressin receptor V1b that promotes ACTH secretion

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