[1P-0129] ICF syndrome proteins CDCA7 and HELLS promote non-homologous end joining through facilitation of Ku80 accumulation at double strand breaks

〇Motoko Unoki¹, Hironori Funabiki², Hiroyuki Sasaki¹ (1.Medical Institute of Bioregulation, Kyushu University, 2.The Rockefeller University)

non-homologous end joining, chromatin remodeling, DNA methylation, centromere, pericentromere