The Molecular Biology Society of Japan

[1P-0129] ICF syndrome proteins CDCA7 and HELLS promote non-homologous end joining through facilitation of Ku80 accumulation at double strand breaks

〇Motoko Unoki1, Hironori Funabiki2, Hiroyuki Sasaki1 (1.Medical Institute of Bioregulation, Kyushu University, 2.The Rockefeller University)

non-homologous end joining, chromatin remodeling, DNA methylation, centromere, pericentromere

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