[2P-1052（2AT26-02）] Neural activity-dependent transcription factor Npas4 is required for neuroprotection and survival after stroke

〇Hiroo Takahashi¹, Ryo Asahina¹, Sei-ichi Yoshihara¹, Akio Tsuboi¹ (1.Lab. for Mol. Biol. of Neural System, Nara Med. Univ.)

ConBio2017

[2P-1053（2AT26-03）] Clodronate attenuates neuropathic and inflammatory pain via inhibition of vesicular nucleotide transporter

〇Yuri Kato^1,2, Miki Hiasa², Reiko Ichikawa³, Nao Hasuzawa⁴, Atsushi Kadowaki⁵, Ken Iwatsuki^3,6, Kazuhiro Shima⁷, Yasuo Endo⁷, Yoshiro Kitahara³, Tsuyoshi Inoue⁵, Masatoshi Nomura⁴, Hiroshi Omote², Yoshinori Moriyama^1,2,8, Takaaki Miyaji¹ (1.Adv. Sci. Res. Cent., Okayama Univ., 2.Dept. of Memb. Biol., Grad. Sch. of Med. Dent. and Pharm. Sci., Okayama Univ., 3.Inst. Innov. Ajinomoto co. Inc., 4.Dept. of Med. and Bio. Sci., Grad. Sch. of Med. Sci., Kyushu Univ., 5.Dept. of Bio. Chem., Grad. Sch. of Med. Dent. and Pharm. Sci., Okayama Univ., 6.Dept. of Nut. Sci. & Food Saf., Tokyo Univ. of Agri., 7.Dept. of Oral Mol. Reg., Grad. Sch. of Dent., Tohoku Univ., 8.Dept. of Bio., Matsumoto Dent. Univ.)

ConBio2017

[2P-1054（2AT26-04）] Neuroprotective Effect of A Novel Ferroptosis Inhibitor

〇Tomonori Hoshino^1,2, Shuichi Matsuzawa^1,2, Ryosuke Takahashi² (1.Drug Screening Incubation Laboratory, Graduate School of Medicine, Kyoto University, Kyoto, Japan, 2.Department of Neurology, Graduate School of Medicine, Kyoto University, Kyoto, Japan)

ConBio2017

[2P-1055（2AT26-05）] Aiming to recover neurological phenotypes of Ehmt1 heterozygous KO mice by re-expression of Ehmt1 after birth

〇Ayumi Yamada¹, Kayako Nishimura¹, Takae Hirasawa², Yoichi Shinkai¹ (1.Cellular Memory Lab., RIKEN, 2.Dept. of Bioscience, Sch. of Sci. and Eng., Teikyo Univ. )

ConBio2017

[2P-1056（2AT26-06）] Neuroprotective role of cystatin C in SOD1-linked amyotrophic lateral sclerosis

〇Seiji Watanabe¹, Okiru Komine¹, Fumito Endo¹, Koji Yamanaka¹ (1.Dept. of Neurosci. Pathobiol., RIEM, Nagoya Univ.)

ConBio2017

[2P-1057（2AT26-07）] Dichotomous effect of systemic overexpression of SQSTM1 on disease onset and post-onset survival in SOD1^H46R mice

〇Shun Mitsui¹, Asako Otomo¹, Masahisa Nozaki¹, Suzuka Ono¹, Kai Sato¹, Ryohei Shirakawa¹, Hiroaki Adachi², Masashi Aoki³, Gen Sobue⁴, Hui-Fang Shang⁵, Shinji Hadano¹ (1.Dept Mol Life Sci, Tokai Univ Sch Med, 2.Dept Neurol, Univ Occup Environ Health Sch Med, 3.Dept Neurol, Tohoku Univ Grad Sch Med, 4.Dept Neurol, Nagoya Univ Grad Sch Med, 5.Dept Neurol, West China Hosp, Sichuan Univ)

ConBio2017

[2P-1058（2AT26-08）] Molecular pathology of dysmyelination caused by defective intracellular cholesterol trafficking

〇Yasushi Enokido¹, Soichiro Kishi¹, Naoko Inamura¹, Masanori Hosokawa¹, Makoto Michikawa², Katsumi Higaki³, Hirohide Takebayashi⁴ (1.Inst. for Dev. Res., Aichi Human. Srv. Cntr., 2.Grad. Sch. of Med. Sci., Nagoya City Univ., 3.Res. Cntr. for Biosci. & Technol., Tottori Univ., 4.Grad. Sch. of Med. & Dntl. Sci., Niigata Univ. )

ConBio2017

[2P-1059（2AT26-09）] ATP maintenance via ATP regulators mitigates pathological phenotypes in a mouse model of Parkinsonʹs disease.

〇Masaki Nakano¹, Hiromi Imamura¹, Norio Sasaoka¹, Masamichi Yamamoto², Norihito Uemura³, Toshiyuki Shudo^1,4, Tomohiro Fuchigami⁴, Ryosuke Takahashi³, Akira Kakizuka¹ (1.Lab. of Func. Bio., Grad. Sch. of Bio., Univ. of Kyoto, 2.Dept. of Nephro., Grad. Sch. of Med., Univ. of Kyoto, 3.Dept. of Neuro., Grad. Sch. of Med., Univ. of Kyoto, 4.Daito Chemix)

ConBio2017

[2P-1060（2AT26-10）] Prenatal neurogenesis induction therapy normalizes brain structure and functions in Down syndrome model mice

〇Akiko Kobayashi¹, Tomonari Awaya¹, Isao Kii¹, Yuto Sumida², Yukiko Okuno³, Keiko Wanezaki¹, Suguru Yoshida², Tomoe Sumida², Haruhisa Inoue⁴, Takamitsu Hosoya², Masatoshi Hagiwara¹ (1.Dept. of Anat. and Dev. Biol., Grad. Sch. of Med., Kyoto Univ., 2.Lab. of Chem. Biosci., Institute of Biomaterials and Bioengineering, Tokyo Med. and Dent. Univ., 3.MRSC, Grad. Sch. of Med., Kyoto Univ., 4.CiRa, Kyoto Univ.)

ConBio2017

[2P-1061（2AT26-11）] Genotype-phenotype relationship of preserved speech variant of Rett syndrome and molecular mechanisms underlying the treatment responsiveness

〇Kenji Orimoto¹, Toyjiro Matsuishi², Kotaro Yuge³ (1.Dept. of Gen. Int. Med., Fureai Higashi-Totsuka Hosp., 2.Res. Ctr. for Ch., Res. Ctr. for RTT, St. Mary's Hosp., 3.Dept. of Pediatr., Sch. of Med., Univ. of Kurume)

ConBio2017